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Protecting against liver damage, such as non-alcoholic fatty liver disease, is currently considered to be important for the prevention of adverse conditions, such as cardiovascular and cancerous diseases. 

Liver damage often occurs in relation to oxidative stress with metabolic disorders, including cellular lipid accumulation. Astaxanthin (3,3′-dihydroxy-β,β-carotene-4,4′dione), a xanthophyll carotenoid, is a candidate for liver protection. 
White paper by John E. Dore, Ph.D. Cyanotech Corporation, Kailua-Kona, Hawaii, 

Astaxanthin has exhibited potent antioxidant, immunomodulating and enzymeinducing properties, all of which suggest a potential role for this carotenoid in the prevention  of cancer. Moreover, its unique structural properties and its lack of prooxidant activity make it a prime candidate for further investigation in this area of human health. 
Astaxanthin, a red pigment that originates in marine algae, is one of nature’s most potent antioxidants. It has numerous other useful properties as well: it is an efficient blocker of ultraviolet radiation, it reduces the impact of glycation, and it decreases inflammatory responses.

Studies are revealing astaxanthin’s remarkable ability to fight the prime causes of aging, not only in the skin, where it was first studied, but in organs and tissues throughout the body. Astaxanthin protects and promotes healthy immune functioning, reduces cancer risks, mitigates the impact of diabesity, protects heart muscle and blood vessels, slows brain aging, and supports eye health.


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Latest Article on Astaxanthin
This study was undertaken to confirm the effect of an astaxanthin-rich Haematococcus pluvialis extract (Astaxanthin) on cognitive function in 96 subjects by a randomised double-blind placebo-controlled study. Healthy middle-aged and elderly subjects who complained of age-related forgetfulness were recruited. ​

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This article concludes that the management of oxidative stress by antioxidant therapies in combination with other inflammatory pathways are the therapeutic weapon to fight against rheumatoid arthritis.
Antioxidants act as free radical scavengers and are thus found to play a significant protective role against oxidative stress in rheumatoid arthritis and in a variety of diseases such as liver cirrhosis, inflammation, atherosclerosis, diabetes, cancer and neurodegenerative diseases. 

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The study demonstrated that because participants with type 2 diabetes often have hypertriglycemia and uncontrolled glucose metabolism, our findings of dual beneficial effects are clinically valuable.

The results offer a novel complementary treatment with potential impacts on diabetic complications without adverse effects.

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This study begins by looking at the current options for people suffering from chronic pain, with important attention to the side effects associated with pain relieving drugs. It will also review Natural Astaxanthin as an anti-inflammatory and as a potential remedy for painful joint and tendon conditions: 

  • ​The first step to examine the mechanism of action for how Astaxanthin works as an anti-inflammatory.
  • Next, we’ll look at how Astaxanthin combats the key marker for inflammation in our bodies: C-reactive protein (CRP). 
  • After this, one-by-one we’ll go through the human clinical trials on inflammatory conditions such as arthritis and tendonitis. 
  • We’ll also list a few of the supporting pre-clinical trials that corroborate the findings in humans to help us round out the body of medical research in this area.
  • Lastly, we’ll discuss the vast differences between Natural Astaxanthin and Synthetic Astaxanthin to ensure our Readers make the right choice when deciding which form to use. 


Oxidative stress induced by hyperglycemia possibly causes the dysfunction of pancreatic β -cells and various forms of tissue damage in patients with diabetes mellitus. Astaxanthin, a carotenoid of marine microalgae, is reported as a strong anti-oxidant inhibiting lipid peroxidation and scavenging reactive oxygen species. The aim of the present study was to examine whether astaxanthin can elicit beneficial effects on the progressive destruction of pancreatic β -cells in db/db mice – a well-known obese model of type 2 diabetes. We used diabetic C57BL/KsJ-db/db mice and db/m for the control. Astaxanthin treatment was started at 6 weeks of age and its effects were evaluated at 10, 14, and 18 weeks of age by non-fasting blood glucose levels, intraperitoneal glucose tolerance test including insulin secretion, and β -cell histology. The non-fasting blood glucose level in db/db mice was significantly higher than that of db/m mice, and the higher level of blood glucose in db/db mice was significantly decreased after treatment with astaxanthin. The ability of islet cells to secrete insulin, as determined by the intraperitoneal glucose tolerance test, was preserved in the astaxanthin-treated group. Histology of the pancreas revealed no significant differences in the β -cell mass between astaxanthin-treated and -untreated db/db mice. In conclusion, these results indicate that astaxanthin can exert beneficial effects in diabetes, with preservation of β-cell function. This finding suggests that anti-oxidants may be potentially useful for reducing glucose toxicity. To read complete study, go to www.valasta.net under Research tab.

Db/db is a mouse that has genetic diabetes.